Link to video
https://rumble.com/vptgyk-small-pox...-now-out-of-africa-into-hong-kong-reported.html
https://www.naturalnews.com/2021-11-18-smallpox-biowar-globalists-prepare-perfect-scheme-to-cover-up-vaccine-deaths-epidemic.html#
https://www.nytimespost.com/questionable-vials-labeled-smallpox-found-in-pennsylvania-lab-prompts-fbi-and-cdc-investigation/?fbclid=IwAR32FfOrso5548F-lhx5HaLjmSx-dBOvRCsVf20DN9RdYWJysVOP_-h1mic
this is the plant that is said to mitigate it's effects. looks like plant in little shop of horrors!
http://www.sarracenia.com/faq/faq5538.html
remedy???
https://www.chemistryworld.com/news/rediscovered-native-american-remedy-kills-poxvirus/3003420.article
got sarcenia purepurea seeds here
https://carnivorousplantnursery.com/
Moreover, treating a purified VACV stock with S. purpurea did not affect replication of the virus, indicating that the extract does not have a direct affect on free virus particles (data not shown)
To determine when S. purpurea treatment was most effective at preventing VACV replication, we examined the ability of S. purpurea to prevent VACV induced CPE when added at various times post-infection. In this assay, cells were infected with VACV and then treated with S. purpurea at the indicated times post-infection. At 6 hpi, the cells were examined for VACV induced CPE. In untreated cells, significant VACV induced CPE, specifically cell rounding, was observed (Fig. 2a). However, cells treated at 0, 15, and 30 minutes post-infection (mpi) with S. purpurea showed no or low levels of CPE. In cells treated at 60 and 120 mpi, substantial CPE was observed These results are in agreement with Fig. 1e that S. purpurea was likely targeting an early component in VACV replication (ie. viral uptake or early viral transcription/translation).
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3302891/
https://www.sciencedirect.com/science/article/abs/pii/S0140673602571476
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